The effects of acoustic deprivation on the recovery of auditory threshold from acoustic trauma
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The effects of acoustic deprivation on the recovery of auditory threshold from acoustic trauma by Michael J. Ruckenstein

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Published by National Library of Canada = Bibliothèque nationale du Canada in Ottawa .
Written in English


Book details:

Edition Notes

SeriesCanadian theses = Thèses canadiennes
The Physical Object
FormatMicroform
Pagination2 microfiches : negative.
ID Numbers
Open LibraryOL14726038M
ISBN 100315742615
OCLC/WorldCa29912329

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  Two weeks after acoustic trauma, ipsilateral (left) CAP thresholds recovered substantially after both durations of acoustic trauma showing a small remaining threshold loss (Fig. 2A, B and C). Mean threshold loss after recovery was significantly different from pre-surgery levels at 12, 20, 22 and 24 kHz after 1 h exposure (Fig.2A) and at 12 Cited by: An auditory-periphery model of the effects of acoustic trauma on auditory nerve responses Ian C. Bruce,a) Murray B. Sachs, and Eric D. Young Center for Hearing Sciences and Department of Biomedical Engineering, Johns Hopkins University School of Medicine, Baltimore, Maryland Recovery of speech perception performance after prolonged auditory deprivation: case study October Journal of the American Academy of Audiology 4(5); discussion Author: Arthur Boothroyd.   In an early human study, the effect of low‐level training acoustic stimulation (70 dB for 6 hr/day for 9 days) on the susceptibility to noise was first assessed in teenage subjects by comparing noise‐induced temporal hearing threshold shifts. 11 During the training period, the subjects listened to music at the 70 dBA prespecified levels.

The main auditory effects include: Acoustic trauma: Sudden hearing damage caused by short burst of extremely loud noise such as a gun shot. Tinnitus: Ringing or buzzing in the ear. Temporary hearing loss: Also known as temporary threshold shift (TTS) which occurs immediately after exposure to a . Groups of chinchillas were exposed to ten ‐dB peak SPL reverberant blast waves from a conventional shock tube at a rate of one blast per minute. Immediately following exposure, the animals were (1) returned to the quiet (approx. 40 dBA) animal colony or (2) placed in a 72‐dB SPL rms broadband noise for 24 h. The broadband noise caused no temporary or permanent hearing losses nor. suggested that auditory evoked responses could be used to estimate rate of recovery and long-term prognosis for recovery, as there was an apparent correlation between electrophysiological auditory results and later behavioral assessment. In cases of closed head injury, the underlying neuropathology is likely to be related to deformation and. loss sensory cell regeneration and functional recovery the effects of acoustic trauma other cochlear injury and death on basilar membrane responses to sound excitotoxicity scientific basis of noise induced hearing loss Posted By Richard Scarry Public Library.

Jos J. Eggermont, in Hearing Loss, Single Auditory Nerve Fiber Responses and Recruitment. Following acoustic trauma, OHC loss is often accompanied by IHC damage (Liberman and Dodds, ), which compromises cochlear transduction and lowers the firing rates of ANFs (Liberman and Kiang, ).As a result, the slopes of their RLFs become shallower on average (Salvi et al., .   Acoustic trauma is an injury to the inner ear that’s often caused by exposure to a high-decibel noise. This injury can occur after exposure to a single, very loud noise or from exposure to.   Hearing Research. 50 () Eisevier HEARES Influence of acoustic deprivation on recovery of hair cells after acoustic trauma N. Fukushima 1, P. White 2 and R.V. Harrison3 ' Department of Otolaryngology University o/Hiroshima, Japan, 1 Department of Otolaryngology University of Toronto. Abstract. Mounting evidence demonstrates the adverse consequences of various forms of auditory deprivation in both animals and humans. The term “auditory deprivation” is a specific form or perversion of auditory input that deviates from what is expected and/or needed for the optimization of auditory function in the organism—be it mouse, or human.